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Medical Hair Restoration Australia

How hair actually grows

Almost every question about hair loss comes back to the growth cycle. Understanding it is the difference between panicking about normal shedding and recognising the point where something has genuinely changed.

Clinically reviewed

Kate Dawes · IAT-registered trichologist · 30+ years in the hair industry · Reviewed · Next review

The follicle

A hair follicle is an organ. It has a blood supply, a nerve supply, its own muscle, and a stem cell population that rebuilds it from scratch several times over a lifetime. There are roughly 100,000 on a human scalp and the number you are born with is the number you get.

At the base sits the dermal papilla, a cluster of cells that supplies the growing hair with nutrients and receives the hormonal signals that determine how long it grows. Nearly everything that matters in hair loss happens here rather than in the visible hair, which is dead keratin by the time you can see it.

Fig. 1The follicle in cross-section
Cross-section of a hair follicle in the skinA vertical section through the skin showing the three layers — epidermis, dermis and subcutis — with a hair follicle running through them. The hair shaft emerges above the surface. Alongside the follicle sit the sebaceous gland and the arrector pili muscle. At the base, the bulb wraps around the dermal papilla, which supplies the blood that keeps the follicle producing hair.EpidermisDermisSubcutisHair shaft — dead keratin.Everything visible is thisSebaceous glandArrector pili muscleFollicle — the living organ,anchored in the dermisDermal papillathe blood supply. What treatmentkeeps, a transplant moves, andscarring alopecia destroysSection through the scalp, not to scale. Around 100,000 of these on an adult head, each cycling independently.

The visible hair is dead keratin; the organ is below the surface. The dermal papilla at the base is the part every treatment route ultimately addresses — medical routes try to keep it working, a transplant physically moves it, and a scarring alopecia destroys it.

  • Cuticle — overlapping scales on the outside; damaged cuticle is why hair feels rough, and has nothing to do with loss.
  • Cortex — the bulk of the shaft, carrying its strength and its pigment.
  • Medulla — a soft core, present in thicker hairs and often absent in fine ones.

The growth cycle

Follicles do not grow continuously and they do not act together. Each one runs its own cycle, which is why hair thins gradually rather than falling out all at once.

Fig. 2The hair growth cycle
The hair growth cycleA timeline of the three phases of the hair growth cycle. Anagen, the growing phase, lasts two to seven years and accounts for about 87 percent of follicles. Catagen, the transition phase, lasts two to three weeks. Telogen, the resting phase, lasts about three months and accounts for 10 to 15 percent of follicles, ending in exogen, when the hair is shed and a new anagen hair begins.87%12%SHARE OF FOLLICLES AT ANY ONE TIMEAnagenGrowing2–7 yearsCatagenTransition2–3 weeksTelogenResting~3 monthsExogenShedA NEW ANAGEN HAIR BEGINS

At any moment roughly 87% of follicles are growing, about 1% are in transition, and 10–15% are resting. Losing 50–100 hairs a day is the resting cohort shedding on schedule. What matters clinically is not the number shed but whether the ratio has shifted.

Anagen is the growing phase and it lasts two to seven years. Its length is what determines how long your hair can get: a three-year anagen gives shoulder-length hair, a seven-year anagen reaches the waist. Catagen is a brief transition of two to three weeks. Telogen is a resting phase of around three months, ending in exogen — the shed itself — as a new anagen hair pushes up beneath.

Miniaturisation — what pattern loss actually is

Pattern hair loss is not follicles dying. It is follicles shrinking. With each cycle the anagen phase gets shorter and the follicle produces a slightly finer, shorter, paler hair. Eventually the hair it makes is a barely visible vellus hair, and eventually the cycle stops.

Fig. 3The hair shaft in cross-section
The three layers of the hair shaft, and what miniaturisation removesTwo cross-sections of a hair shaft. The left, a terminal hair, shows three concentric layers: the cuticle on the outside, the thick cortex beneath it, and the medulla at the centre. The right, a miniaturised hair, is drawn much smaller: the cortex has thinned and the medulla is absent, which is why miniaturised hair is finer, weaker and lighter in colour.Terminal hair~70–100 µm acrossMiniaturisedcortex thinned, no medullaCuticle — overlapping scales,the protective surfaceCortex — the bulk, strengthand pigment of the hairMedulla — the soft core,absent in fine hairSame follicle, different output. Miniaturisation shrinks the cortex cycle by cycle — the hair is not falling out so much as being rebuilt smaller.

What miniaturisation removes, layer by layer. The cortex — the strength and pigment of the hair — thins first, and the medulla disappears from fine hair entirely. The same follicle is producing both hairs shown; only its output has changed.

This is why treatment is time-sensitive in a way that is genuinely different from most cosmetic decisions. A miniaturised follicle is still a working follicle and can often be helped. Once it has been dormant for years, it cannot. The window is measured in years, but it is a window.

It is also why the first visible sign is usually a change in texture rather than a change in coverage. Hair that has become finer and will not hold a style is the same process, six to eighteen months earlier.

What is normal

Fifty to a hundred hairs a day is normal. Shedding more after a wash is normal, because you are collecting two or three days at once. Seasonal variation is normal — telogen rates rise for most people in late autumn.

What is worth attention: a sustained increase lasting more than three months, a visible change in part width, hair that has become noticeably finer, or shedding accompanied by scalp symptoms such as itching, burning or tenderness.

Common questions

How many hairs a day is too many?

The number is less useful than the trend. Fifty to a hundred is normal, and most people cannot count accurately anyway. A sustained change over three months, or a visible change in part width, is a better signal than any single day.

Does cutting hair make it grow back thicker?

No. Growth happens at the follicle, below the skin. Cutting removes the tapered end, which makes the remaining shaft feel coarser at the tip, but it changes nothing about rate or diameter.

Does wearing hats cause hair loss?

No, unless the hat is tight enough to cause traction over years. Follicles get oxygen from blood supply, not from air.

Why does hair stop growing at a certain length?

Because the anagen phase has a genetically determined length. When it ends the hair stops growing regardless of how it is treated. Length potential is set by cycle duration, not by care.

Can a follicle come back once it has stopped?

A miniaturised follicle that is still cycling can often produce a better hair with treatment. A follicle that has been dormant for years, or destroyed by a scarring process, does not return. This distinction is the single most important one in hair loss.

Is grey hair related to hair loss?

They are separate processes. Greying is pigment cell exhaustion in the follicle; loss is a cycling change. They often arrive around the same age, which is why they get conflated.

References

What an assessment establishes

  1. 1.
    Which process is causing the loss

    Pattern loss, telogen effluvium, traction and scarring alopecia look similar in a mirror and behave nothing alike. The examination separates them, because the answer decides which routes are worth discussing at all.

  2. 2.
    Whether it is stable or still moving

    Loss that is still progressing changes what any treatment can hold. It is the difference between planning around a fixed area and planning around one that will keep widening, and it is judged from history and examination rather than a single photograph.

  3. 3.
    Which routes are available to you, and which are not

    Donor supply, scalp condition, skin type and how the loss is behaving each rule options in or out. You leave knowing what is on the table and what is not, without a price attached to the conversation.

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